Myth: Alternative nicotine products are as dangerous as smoking: Difference between revisions

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===2019: [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6592370/ Electronic cigarettes and cardiovascular health: what do we know so far?]===  
===2019: [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6592370/ Electronic cigarettes and cardiovascular health: what do we know so far?]===  
*Though they may not be as harmless as previously proposed, it seems likely that on the spectrum of tobacco products, ECs are less harmful than TCs, and there is increasing evidence that ECs may help promote TC cessation. As such, ECs may be helpful for risk reduction
*Though they may not be as harmless as previously proposed, it seems likely that on the spectrum of tobacco products, ECs are less harmful than TCs, and there is increasing evidence that ECs may help promote TC cessation. As such, ECs may be helpful for risk reduction.
*Citation: MacDonald A, Middlekauff HR. Electronic cigarettes and cardiovascular health: what do we know so far? Vasc Health Risk Manag. 2019 Jun 21;15:159-174. doi: 10.2147/VHRM.S175970. PMID: 31417268; PMCID: PMC6592370.
 
===2017: [https://www.sciencedirect.com/science/article/pii/S0378427417302266 A comparative assessment of e-cigarette aerosols and cigarette smoke on in vitro endothelial cell migration]===
===2017: [https://www.sciencedirect.com/science/article/pii/S0378427417302266 A comparative assessment of e-cigarette aerosols and cigarette smoke on in vitro endothelial cell migration]===
*Exposure to 3R4F extract (20h) induced concentration-dependent inhibition of endothelial cell migration, with complete inhibition at concentrations >20%. E-cigarette extracts did not inhibit migration, even at double the 3R4F extract nicotine concentration, allowing cells to migrate into the wounded area. Our data demonstrate that e-cigarettes do not induce the inhibition of endothelial cell migration in vitro when compared to 3R4F.
*Exposure to 3R4F extract (20h) induced concentration-dependent inhibition of endothelial cell migration, with complete inhibition at concentrations >20%. E-cigarette extracts did not inhibit migration, even at double the 3R4F extract nicotine concentration, allowing cells to migrate into the wounded area. Our data demonstrate that e-cigarettes do not induce the inhibition of endothelial cell migration in vitro when compared to 3R4F.