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**“The study has unclear relevance for human vapers. Rodents were exposed to what are for them huge concentrations of chemicals that bear no resemblance to human exposure from vaping. Several animals in fact died during these exposures. The authors assigned the effects they observed to a carcinogen NNK – but NNK has been measured before in human vapers, and it is known that exposure from vaping is either negligible or none.” (Hajek) | **“The study has unclear relevance for human vapers. Rodents were exposed to what are for them huge concentrations of chemicals that bear no resemblance to human exposure from vaping. Several animals in fact died during these exposures. The authors assigned the effects they observed to a carcinogen NNK – but NNK has been measured before in human vapers, and it is known that exposure from vaping is either negligible or none.” (Hajek) | ||
*Referring to: Tang MS, Wu XR, Lee HW, Xia Y, Deng FM, Moreira AL, Chen LC, Huang WC, Lepor H. Electronic-cigarette smoke induces lung adenocarcinoma and bladder urothelial hyperplasia in mice. Proc Natl Acad Sci U S A. 2019 Oct 22;116(43):21727-21731. doi: 10.1073/pnas.1911321116. Epub 2019 Oct 7. Erratum in: Proc Natl Acad Sci U S A. 2019 Nov 5;116(45):22884. PMID: 31591243; PMCID: PMC6815158. | *Referring to: Tang MS, Wu XR, Lee HW, Xia Y, Deng FM, Moreira AL, Chen LC, Huang WC, Lepor H. Electronic-cigarette smoke induces lung adenocarcinoma and bladder urothelial hyperplasia in mice. Proc Natl Acad Sci U S A. 2019 Oct 22;116(43):21727-21731. doi: 10.1073/pnas.1911321116. Epub 2019 Oct 7. Erratum in: Proc Natl Acad Sci U S A. 2019 Nov 5;116(45):22884. PMID: 31591243; PMCID: PMC6815158. | ||
===2017: [https://pubpeer.com/publications/41F6EA57D0803EEE9DF65162DF0097 Electronic Cigarette Use in US Adults at Risk for or with COPD: Analysis from Two Observational Cohorts]=== | |||
*"It is literally true that they "find no evidence...", but that is because this study is completely ill-suited to drawing any policy conclusions about e-cigarettes and COPD. Despite hinting at the limitations of cross-sectional data, the authors draw a negative-sounding conclusion without addressing the key question of how respiratory health changes for a given smoker who uses e-cigarettes to quit or cut down once they are ill from smoking or as a way of preventing COPD." (Bates) | |||
*Referring to: Bowler RP, Hansel NN, Jacobson S, Graham Barr R, Make BJ, Han MK, O'Neal WK, Oelsner EC, Casaburi R, Barjaktarevic I, Cooper C, Foreman M, Wise RA, DeMeo DL, Silverman EK, Bailey W, Harrington KF, Woodruff PG, Drummond MB; for COPDGene and SPIROMICS Investigators. Electronic Cigarette Use in US Adults at Risk for or with COPD: Analysis from Two Observational Cohorts. J Gen Intern Med. 2017 Dec;32(12):1315-1322. doi: 10.1007/s11606-017-4150-7. Epub 2017 Sep 7. PMID: 28884423; PMCID: PMC5698219. | |||
===2017: [https://pubpeer.com/publications/6E2711F55673ADF831D636E2D701B9 "Juice Monsters": Sub-Ohm Vaping and Toxic Volatile Aldehyde Emissions]=== | ===2017: [https://pubpeer.com/publications/6E2711F55673ADF831D636E2D701B9 "Juice Monsters": Sub-Ohm Vaping and Toxic Volatile Aldehyde Emissions]=== | ||
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